Preliminary experiments show conolidine may well inhibit particular ion channels, decreasing neuronal excitability and restricting ache signals. This mechanism is especially related in neuropathic pain, in which irregular signaling will cause persistent distress. Additionally, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to ache perception. Computatio... https://roberti681dyu4.webdesign96.com/profile