Gastrointestinal (GI): NSAIDs that block COX-one activity have correlations with GI complications a result of the inhibition with the synthesis of gastroprotective brokers such as PGE2 as well as other prostaglandins. This inhibition brings about diminished mucin generation by gastric epithelial cells, much less bicarbonate secretion, and fewer epithelial cell https://aldousb680bfh6.ltfblog.com/profile